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We observed that it was not necessary to induce the tac promoter to establish sufficient AP synthesis for our purpose, and so we did not do so, preferring steady AP production

It also induces AMPK phosphorylation on threonine residue 172, which inhibits the nuclear translocation of sterol response element binding protein 1 and consequently suppresses the transcription of its direct target gene, BCAT2 and enhances the ferroptosis susceptibility of HCC cells (73)